4,4-dimethylcholesta-8,11,24-trienol

4,4-dimethylcholesta-8,11,24-trienol is a lipid of Sterol Lipids (ST) class. The involved functions are known as Crossbreeding.

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Introduction

To understand associated biological information of 4,4-dimethylcholesta-8,11,24-trienol, we collected biological information of abnormalities, associated pathways, cellular/molecular locations, biological functions, related genes/proteins, lipids and common seen animal/experimental models with organized paragraphs from literatures.

What diseases are associated with 4,4-dimethylcholesta-8,11,24-trienol?

There are no associated biomedical information in the current reference collection.

No disease MeSH terms mapped to the current reference collection.

PubChem Associated disorders and diseases

What pathways are associated with 4,4-dimethylcholesta-8,11,24-trienol

There are no associated biomedical information in the current reference collection.

PubChem Biomolecular Interactions and Pathways

Link to PubChem Biomolecular Interactions and Pathways

What cellular locations are associated with 4,4-dimethylcholesta-8,11,24-trienol?

There are no associated biomedical information in the current reference collection.

What functions are associated with 4,4-dimethylcholesta-8,11,24-trienol?


Related references are published most in these journals:

Function Cross reference Weighted score Related literatures

What lipids are associated with 4,4-dimethylcholesta-8,11,24-trienol?

There are no associated biomedical information in the current reference collection.

What genes are associated with 4,4-dimethylcholesta-8,11,24-trienol?

There are no associated biomedical information in the current reference collection.

What common seen animal models are associated with 4,4-dimethylcholesta-8,11,24-trienol?

There are no associated biomedical information in the current reference collection.

NCBI Entrez Crosslinks

All references with 4,4-dimethylcholesta-8,11,24-trienol

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Authors Title Published Journal PubMed Link
Gatticchi L et al. Selected cholesterol biosynthesis inhibitors produce accumulation of the intermediate FF-MAS that targets nucleus and activates LXRα in HepG2 cells. 2017 Biochim. Biophys. Acta pmid:28499814
Fakheri RJ and Javitt NB Autoregulation of cholesterol synthesis: physiologic and pathophysiologic consequences. 2011 Steroids pmid:20951718
Wang F et al. Gonadotropin-regulated expressions of lanosterol 14alpha-demethylase, sterol Delta14-reductase and C-4 sterol methyl oxidase contribute to the accumulation of meiosis-activating sterol in rabbit gonads. 2010 Prostaglandins Other Lipid Mediat. pmid:20193772
Cavilla JL et al. Human immature oocytes grow during culture for IVM. 2008 Hum. Reprod. pmid:17932084
Grøndahl C Oocyte maturation. Basic and clinical aspects of in vitro maturation (IVM) with special emphasis of the role of FF-MAS. 2008 Dan Med Bull pmid:18321442
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Tsafriri A and Motola S Are steroids dispensable for meiotic resumption in mammals? 2007 Trends Endocrinol. Metab. pmid:17826173
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Smitz J et al. Principal findings from a multicenter trial investigating the safety of follicular-fluid meiosis-activating sterol for in vitro maturation of human cumulus-enclosed oocytes. 2007 Fertil. Steril. pmid:17198705
Bokal EV et al. Follicular sterol composition in gonadotrophin stimulated women with polycystic ovarian syndrome. 2006 Mol. Cell. Endocrinol. pmid:16516374