CYTOCHALASIN B

CYTOCHALASIN B is a lipid of Polyketides (PK) class. Cytochalasin b is associated with abnormalities such as Renal tubular disorder and Chagas Disease. The involved functions are known as Membrane Protein Traffic, inhibitors, Metabolic Inhibition, Biochemical Pathway and Increased Sensitivy. Cytochalasin b often locates in Cytoplasmic matrix, Plasma membrane, Microtubules, Extracellular and Protoplasm. The associated genes with CYTOCHALASIN B are SLC2A2 gene, PFDN5 gene, SLC2A1 gene, OMG gene and SPEN gene. The related lipids are Steroids, Lipopolysaccharides and Liposomes. The related experimental models are Xenograft Model.

Cross Reference

Introduction

To understand associated biological information of CYTOCHALASIN B, we collected biological information of abnormalities, associated pathways, cellular/molecular locations, biological functions, related genes/proteins, lipids and common seen animal/experimental models with organized paragraphs from literatures.

What diseases are associated with CYTOCHALASIN B?

CYTOCHALASIN B is suspected in Renal tubular disorder, Chagas Disease and other diseases in descending order of the highest number of associated sentences.

Related references are mostly published in these journals:

Disease Cross reference Weighted score Related literature
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Possible diseases from mapped MeSH terms on references

We collected disease MeSH terms mapped to the references associated with CYTOCHALASIN B

MeSH term MeSH ID Detail
Hemolysis D006461 131 associated lipids
Tuberculosis D014376 20 associated lipids
Uremia D014511 33 associated lipids
Diabetes Mellitus D003920 90 associated lipids
Adenocarcinoma D000230 166 associated lipids
Dermatitis, Contact D003877 59 associated lipids
Lupus Erythematosus, Systemic D008180 43 associated lipids
Lung Neoplasms D008175 171 associated lipids
Wounds and Injuries D014947 20 associated lipids
Adenoma, Islet Cell D007516 7 associated lipids
Insulinoma D007340 28 associated lipids
Pancreatic Neoplasms D010190 77 associated lipids
Inflammation D007249 119 associated lipids
Colonic Neoplasms D003110 161 associated lipids
Diabetes Mellitus, Type 2 D003924 87 associated lipids
Diabetic Nephropathies D003928 39 associated lipids
Diabetes Mellitus, Experimental D003921 85 associated lipids
Mammary Neoplasms, Experimental D008325 67 associated lipids
Body Weight D001835 333 associated lipids
Foreign-Body Reaction D005549 10 associated lipids
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PubChem Associated disorders and diseases

What pathways are associated with CYTOCHALASIN B

There are no associated biomedical information in the current reference collection.

PubChem Biomolecular Interactions and Pathways

Link to PubChem Biomolecular Interactions and Pathways

What cellular locations are associated with CYTOCHALASIN B?

Related references are published most in these journals:

Location Cross reference Weighted score Related literatures
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What functions are associated with CYTOCHALASIN B?


Related references are published most in these journals:

Function Cross reference Weighted score Related literatures

What lipids are associated with CYTOCHALASIN B?

Related references are published most in these journals:

Lipid concept Cross reference Weighted score Related literatures
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What genes are associated with CYTOCHALASIN B?

Related references are published most in these journals:


Gene Cross reference Weighted score Related literatures

What common seen animal models are associated with CYTOCHALASIN B?

Xenograft Model

Xenograft Model are used in the study 'Endofacial competitive inhibition of the glucose transporter 1 activity by gossypol.' (Pérez A et al., 2009).

Related references are published most in these journals:

Model Cross reference Weighted score Related literatures
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NCBI Entrez Crosslinks

All references with CYTOCHALASIN B

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Authors Title Published Journal PubMed Link
Kole MH et al. Action potential generation requires a high sodium channel density in the axon initial segment. 2008 Nat. Neurosci. pmid:18204443
Koltsova SV et al. Vascular smooth muscle contraction evoked by cell volume modulation: role of the cytoskeleton network. 2008 Cell. Physiol. Biochem. pmid:18209469
Chen JJ et al. Neolignans, a coumarinolignan, lignan derivatives, and a chromene: anti-inflammatory constituents from Zanthoxylum avicennae. 2008 J. Nat. Prod. pmid:18211005
Chen DC et al. Eugenol inhibited the antimicrobial functions of neutrophils. 2008 J Endod pmid:18215676
Corrêa JR et al. Transferrin uptake in Trypanosoma cruzi is impaired by interference on cytostome-associated cytoskeleton elements and stability of membrane cholesterol, but not by obstruction of clathrin-dependent endocytosis. 2008 Exp. Parasitol. pmid:18234197
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Terracciano S et al. Synthetic and pharmacological studies on new simplified analogues of the potent actin-targeting Jaspamide. 2008 Bioorg. Med. Chem. pmid:18508272
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Alexandrova ML et al. Inhibitory and enhancing effects of piroxicam on whole blood chemiluminescence. 2007 Mar-Apr Luminescence pmid:17089362
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Formigli L et al. Cytoskeleton/stretch-activated ion channel interaction regulates myogenic differentiation of skeletal myoblasts. 2007 J. Cell. Physiol. pmid:17295211
Neel NF et al. RhoB plays an essential role in CXCR2 sorting decisions. 2007 J. Cell. Sci. pmid:17405813
Wojda A et al. Effects of age and gender on micronucleus and chromosome nondisjunction frequencies in centenarians and younger subjects. 2007 Mutagenesis pmid:17284771
Bosco D et al. Differential expression of E-cadherin at the surface of rat beta-cells as a marker of functional heterogeneity. 2007 J. Endocrinol. pmid:17592017
Sandoval M et al. p53 response to arsenic exposure in epithelial cells: protein kinase B/Akt involvement. 2007 Toxicol. Sci. pmid:17567589
Polakof S et al. In vitro evidences for glucosensing capacity and mechanisms in hypothalamus, hindbrain, and Brockmann bodies of rainbow trout. 2007 Am. J. Physiol. Regul. Integr. Comp. Physiol. pmid:17567722
Cheng WM et al. Effect of different parthenogenetic activation methods on the developmental competence of in vitro matured porcine oocytes. 2007 Anim. Biotechnol. pmid:17453653
Alvarado-Vásquez N et al. HUVECs from newborns with a strong family history of diabetes show diminished ROS synthesis in the presence of high glucose concentrations. 2007 Diabetes Metab. Res. Rev. pmid:16810702