CYTOCHALASIN B is a lipid of Polyketides (PK) class. Cytochalasin b is associated with abnormalities such as Renal tubular disorder and Chagas Disease. The involved functions are known as Membrane Protein Traffic, inhibitors, Metabolic Inhibition, Biochemical Pathway and Increased Sensitivy. Cytochalasin b often locates in Cytoplasmic matrix, Plasma membrane, Microtubules, Extracellular and Protoplasm. The associated genes with CYTOCHALASIN B are SLC2A2 gene, PFDN5 gene, SLC2A1 gene, OMG gene and SPEN gene. The related lipids are Steroids, Lipopolysaccharides and Liposomes. The related experimental models are Xenograft Model.
To understand associated biological information of CYTOCHALASIN B, we collected biological information of abnormalities, associated pathways, cellular/molecular locations, biological functions, related genes/proteins, lipids and common seen animal/experimental models with organized paragraphs from literatures.
CYTOCHALASIN B is suspected in Renal tubular disorder, Chagas Disease and other diseases in descending order of the highest number of associated sentences.
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We collected disease MeSH terms mapped to the references associated with CYTOCHALASIN B
There are no associated biomedical information in the current reference collection.
Associated locations are in red color. Not associated locations are in black.
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Function | Cross reference | Weighted score | Related literatures |
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Lipid concept | Cross reference | Weighted score | Related literatures |
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Gene | Cross reference | Weighted score | Related literatures |
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Xenograft Model are used in the study 'Endofacial competitive inhibition of the glucose transporter 1 activity by gossypol.' (Pérez A et al., 2009).
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Authors | Title | Published | Journal | PubMed Link |
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Sayós J et al. | Regulation of nitrobenzylthioninosine-sensitive adenosine uptake by cultured kidney cells. | 1994 | Am. J. Physiol. | pmid:7977779 |
Mullin JM et al. | Isolation of mutant renal (LLC-PK1) epithelia defective in basolateral, Na(+)-independent glucose transport. | 1989 | Am. J. Physiol. | pmid:2603953 |
Goodyear LJ et al. | Glucose transporter number, activity, and isoform content in plasma membranes of red and white skeletal muscle. | 1991 | Am. J. Physiol. | pmid:1951679 |
Goodyear LJ et al. | Exercise-induced translocation of skeletal muscle glucose transporters. | 1991 | Am. J. Physiol. | pmid:1662910 |
Fürnsinn C et al. | More marked stimulation by lithium than insulin of the glycogenic pathway in rat skeletal muscle. | 1997 | Am. J. Physiol. | pmid:9316440 |
Ehrhardt RA and Bell AW | Developmental increases in glucose transporter concentration in the sheep placenta. | 1997 | Am. J. Physiol. | pmid:9321896 |
Lichtman SN et al. | Endocytosis and Ca2+ are required for endotoxin-stimulated TNF-alpha release by rat Kupffer cells. | 1996 | Am. J. Physiol. | pmid:8944708 |
Henquin JC and Lambert AE | Bicarbonate modulation of glucose-9nduced biphasic insulin release by rat islets. | 1976 | Am. J. Physiol. | pmid:788525 |
Halm DR et al. | Selective stimulation of epithelial cells in colonic crypts: relation to active chloride secretion. | 1995 | Am. J. Physiol. | pmid:7485463 |
Jacobs DB et al. | Alterations of glucose transporter systems in insulin-resistant uremic rats. | 1989 | Am. J. Physiol. | pmid:2669514 |