palmitic acid

palmitic acid is a lipid of Fatty Acyls (FA) class. The involved functions are known as Apoptosis, Synthesis, inhibitors, Oxidation and targeting. Palmitic acid often locates in Extracellular, Muscle, Protoplasm, Body tissue and Blood. The related lipids are Palmitates, Sodium Palmitate and saturated fat.

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Introduction

To understand associated biological information of palmitic acid, we collected biological information of abnormalities, associated pathways, cellular/molecular locations, biological functions, related genes/proteins, lipids and common seen animal/experimental models with organized paragraphs from literatures.

What diseases are associated with palmitic acid?

There are no associated biomedical information in the current reference collection.

No disease MeSH terms mapped to the current reference collection.

PubChem Associated disorders and diseases

What pathways are associated with palmitic acid

There are no associated biomedical information in the current reference collection.

PubChem Biomolecular Interactions and Pathways

Link to PubChem Biomolecular Interactions and Pathways

What cellular locations are associated with palmitic acid?

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What functions are associated with palmitic acid?


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What lipids are associated with palmitic acid?

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What genes are associated with palmitic acid?

There are no associated biomedical information in the current reference collection.

What common seen animal models are associated with palmitic acid?

There are no associated biomedical information in the current reference collection.

NCBI Entrez Crosslinks

All references with palmitic acid

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Authors Title Published Journal PubMed Link
Saeed NM et al. Anti-inflammatory activity of methyl palmitate and ethyl palmitate in different experimental rat models. 2012 Toxicol. Appl. Pharmacol. pmid:22842335
Luo Y et al. Cyclosporine A and palmitic acid treatment synergistically induce cytotoxicity in HepG2 cells. 2012 Toxicol. Appl. Pharmacol. pmid:22521608
Chen X et al. An anti-inflammatory chalcone derivative prevents heart and kidney from hyperlipidemia-induced injuries by attenuating inflammation. 2018 Toxicol. Appl. Pharmacol. pmid:29128402
Vesterdal LK et al. Accumulation of lipids and oxidatively damaged DNA in hepatocytes exposed to particles. 2014 Toxicol. Appl. Pharmacol. pmid:24121055
Wang S et al. Zinc deficiency exacerbates while zinc supplement attenuates cardiac hypertrophy in high-fat diet-induced obese mice through modulating p38 MAPK-dependent signaling. 2016 Toxicol. Lett. pmid:27346292
Wilson R et al. Uptake and vascular transport of ingested aflatoxin. 1985 Toxicol. Lett. pmid:3937298
Mitra A et al. Inhibition of human term placental and fetal liver glutathione-S-transferases by fatty acids and fatty acid esters. 1992 Toxicol. Lett. pmid:1595087
Fratantonio D et al. Palmitate-induced endothelial dysfunction is attenuated by cyanidin-3-O-glucoside through modulation of Nrf2/Bach1 and NF-κB pathways. 2015 Toxicol. Lett. pmid:26422990
Montaño M et al. New approaches to assess the transthyretin binding capacity of bioactivated thyroid hormone disruptors. 2012 Toxicol. Sci. pmid:22859314
Raucy JL et al. Regulation of CYP2E1 by ethanol and palmitic acid and CYP4A11 by clofibrate in primary cultures of human hepatocytes. 2004 Toxicol. Sci. pmid:15056802