Myrcene

Myrcene is a lipid of Prenol Lipids (PR) class. Myrcene is associated with abnormalities such as Hypomenorrhea, abnormal fragmented structure, Nephrosis, Renal tubular disorder and Kidney Diseases. The involved functions are known as Anabolism, Gene Expression, Protein Biosynthesis, Mutation and Selection, Genetic. Myrcene often locates in Plastids, Proboscis, Body tissue, Clone and soluble. The associated genes with Myrcene are TTPA gene, monoterpene synthase, Genome, 4S-limonene synthase and Homologous Gene. The related lipids are Pinene, Octanols, Membrane Lipids and Fatty Acids, Unsaturated.

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Introduction

To understand associated biological information of Myrcene, we collected biological information of abnormalities, associated pathways, cellular/molecular locations, biological functions, related genes/proteins, lipids and common seen animal/experimental models with organized paragraphs from literatures.

What diseases are associated with Myrcene?

Myrcene is suspected in Nephrosis, Kidney Diseases, Dehydration, Hypomenorrhea, Renal tubular disorder and other diseases in descending order of the highest number of associated sentences.

Related references are mostly published in these journals:

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No disease MeSH terms mapped to the current reference collection.

PubChem Associated disorders and diseases

What pathways are associated with Myrcene

There are no associated biomedical information in the current reference collection.

PubChem Biomolecular Interactions and Pathways

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What cellular locations are associated with Myrcene?

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What functions are associated with Myrcene?


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What lipids are associated with Myrcene?

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What genes are associated with Myrcene?

Related references are published most in these journals:


Gene Cross reference Weighted score Related literatures

What common seen animal models are associated with Myrcene?

There are no associated biomedical information in the current reference collection.

NCBI Entrez Crosslinks

All references with Myrcene

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Authors Title Published Journal PubMed Link
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