sphingosine

sphingosine is a lipid of Sphingolipids (SP) class.

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There are no associated biomedical information in the current reference collection.

Current reference collection contains 26061 references associated with sphingosine in LipidPedia. Due to lack of full text of references or no associated biomedical terms are recognized in our current text-mining method, we cannot extract any biomedical terms related to diseases, pathways, locations, functions, genes, lipids, and animal models from the associated reference collection.

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Here are additional resources we collected from PubChem and MeSH for sphingosine

PubChem Biomolecular Interactions and Pathways

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All references with sphingosine

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Authors Title Published Journal PubMed Link
Contaifer D et al. Unsupervised analysis of combined lipid and coagulation data reveals coagulopathy subtypes among dialysis patients. 2017 J. Lipid Res. pmid:27993949
Sánchez DI et al. Melatonin prevents deregulation of the sphingosine kinase/sphingosine 1-phosphate signaling pathway in a mouse model of diethylnitrosamine-induced hepatocellular carcinoma. 2017 J. Pineal Res. pmid:27696512
Syed SN et al. S1P Provokes Tumor Lymphangiogenesis via Macrophage-Derived Mediators Such as IL-1 or Lipocalin-2. 2017 Mediators Inflamm. pmid:28804221
Doan NB et al. Acid ceramidase confers radioresistance to glioblastoma cells. 2017 Oncol. Rep. pmid:28765947
Reinhard NR et al. The balance between Gα-Cdc42/Rac and Gα/-RhoA pathways determines endothelial barrier regulation by sphingosine-1-phosphate. 2017 Mol. Biol. Cell pmid:28954861
Dany M Sphingosine metabolism as a therapeutic target in cutaneous melanoma. 2017 Transl Res pmid:28528915
Chen Y et al. Atherosclerotic dyslipidemia revealed by plasma lipidomics on ApoE mice fed a high-fat diet. 2017 Atherosclerosis pmid:28527370
Janecke AR et al. Deficiency of the sphingosine-1-phosphate lyase SGPL1 is associated with congenital nephrotic syndrome and congenital adrenal calcifications. 2017 Hum. Mutat. pmid:28181337
Werth S et al. Obesity-stimulated aldosterone release is not related to an S1P-dependent mechanism. 2017 J. Endocrinol. pmid:28970286
Ren K et al. ApoA-I/SR-BI modulates S1P/S1PR2-mediated inflammation through the PI3K/Akt signaling pathway in HUVECs. 2017 J. Physiol. Biochem. pmid:28181168