18alpha-glycyrrhetinic acid

18alpha-glycyrrhetinic acid is a lipid of Prenol Lipids (PR) class. 18alpha-glycyrrhetinic acid is associated with abnormalities such as Wiskott-Aldrich Syndrome. The involved functions are known as inhibitors, salivary gland development and branching morphogenesis.

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Introduction

To understand associated biological information of 18alpha-glycyrrhetinic acid, we collected biological information of abnormalities, associated pathways, cellular/molecular locations, biological functions, related genes/proteins, lipids and common seen animal/experimental models with organized paragraphs from literatures.

What diseases are associated with 18alpha-glycyrrhetinic acid?

18alpha-glycyrrhetinic acid is suspected in and other diseases in descending order of the highest number of associated sentences.

Related references are mostly published in these journals:

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No disease MeSH terms mapped to the current reference collection.

PubChem Associated disorders and diseases

What pathways are associated with 18alpha-glycyrrhetinic acid

There are no associated biomedical information in the current reference collection.

PubChem Biomolecular Interactions and Pathways

Link to PubChem Biomolecular Interactions and Pathways

What cellular locations are associated with 18alpha-glycyrrhetinic acid?

There are no associated biomedical information in the current reference collection.

What functions are associated with 18alpha-glycyrrhetinic acid?


Related references are published most in these journals:

Function Cross reference Weighted score Related literatures

What lipids are associated with 18alpha-glycyrrhetinic acid?

There are no associated biomedical information in the current reference collection.

What genes are associated with 18alpha-glycyrrhetinic acid?

There are no associated biomedical information in the current reference collection.

What common seen animal models are associated with 18alpha-glycyrrhetinic acid?

There are no associated biomedical information in the current reference collection.

NCBI Entrez Crosslinks

All references with 18alpha-glycyrrhetinic acid

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Authors Title Published Journal PubMed Link
Luksha L et al. Endothelium-derived hyperpolarizing factor in preeclampsia: heterogeneous contribution, mechanisms, and morphological prerequisites. 2008 Am. J. Physiol. Regul. Integr. Comp. Physiol. pmid:18032472
Yamanouchi K et al. 18alpha-glycyrrhetinic acid induces phenotypic changes of skeletal muscle cells to enter adipogenesis. 2007 Cell. Physiol. Biochem. pmid:17982260
Yamamoto Y et al. Role of gap junctions and protein kinase A during the development of oocyte maturational competence in Ayu (Plecoglossus altivelis). 2008 Gen. Comp. Endocrinol. pmid:17964574
Lang RJ et al. Spontaneous electrical and Ca2+ signals in typical and atypical smooth muscle cells and interstitial cell of Cajal-like cells of mouse renal pelvis. 2007 J. Physiol. (Lond.) pmid:17656432
Ikeda Y et al. Role of gap junctions in spontaneous activity of the rat bladder. 2007 Am. J. Physiol. Renal Physiol. pmid:17581924
Guan BC et al. Blockade of gap junction coupling by glycyrrhetinic acids in guinea pig cochlear artery: a whole-cell voltage- and current-clamp study. 2007 Br. J. Pharmacol. pmid:17572704
Lin D et al. Protein kinase C gamma mutations in the C1B domain cause caspase-3-linked apoptosis in lens epithelial cells through gap junctions. 2007 Exp. Eye Res. pmid:17493614
Kamioka H et al. Primary cultures of chick osteocytes retain functional gap junctions between osteocytes and between osteocytes and osteoblasts. 2007 Microsc. Microanal. pmid:17367550
Kamijo M et al. The function of connexin 43 on the differentiation of rat bone marrow cells in culture. 2006 Biomed. Res. pmid:17213685
Miyazato M et al. A gap junction blocker inhibits isolated whole bladder activity in normal rats and rats with partial bladder outlet obstruction. 2006 Biomed. Res. pmid:17099284