Elaidamide

Elaidamide is a lipid of Fatty Acyls (FA) class. Elaidamide is associated with abnormalities such as Wiskott-Aldrich Syndrome. The involved functions are known as inhibitors, salivary gland development and branching morphogenesis.

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Introduction

To understand associated biological information of Elaidamide, we collected biological information of abnormalities, associated pathways, cellular/molecular locations, biological functions, related genes/proteins, lipids and common seen animal/experimental models with organized paragraphs from literatures.

What diseases are associated with Elaidamide?

Elaidamide is suspected in and other diseases in descending order of the highest number of associated sentences.

Related references are mostly published in these journals:

Disease Cross reference Weighted score Related literature
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Possible diseases from mapped MeSH terms on references

We collected disease MeSH terms mapped to the references associated with Elaidamide

PubChem Associated disorders and diseases

What pathways are associated with Elaidamide

There are no associated biomedical information in the current reference collection.

PubChem Biomolecular Interactions and Pathways

Link to PubChem Biomolecular Interactions and Pathways

What cellular locations are associated with Elaidamide?

There are no associated biomedical information in the current reference collection.

What functions are associated with Elaidamide?


Related references are published most in these journals:

Function Cross reference Weighted score Related literatures

What lipids are associated with Elaidamide?

There are no associated biomedical information in the current reference collection.

What genes are associated with Elaidamide?

There are no associated biomedical information in the current reference collection.

What common seen animal models are associated with Elaidamide?

There are no associated biomedical information in the current reference collection.

NCBI Entrez Crosslinks

All references with Elaidamide

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Authors Title Published Journal PubMed Link
Gobbi M et al. Oleamide-mediated sleep induction does not depend on perturbation of membrane homeoviscosity. 1999 FEBS Lett. pmid:10606738
Ritenour-Rodgers KJ et al. Induction of peptidylglycine alpha-amidating monooxygenase in N(18)TG(2) cells: a model for studying oleamide biosynthesis. 2000 Biochem. Biophys. Res. Commun. pmid:10631094
Verdon B et al. Stereoselective modulatory actions of oleamide on GABA(A) receptors and voltage-gated Na(+) channels in vitro: a putative endogenous ligand for depressant drug sites in CNS. 2000 Br. J. Pharmacol. pmid:10694234
Quist AP et al. Physiological role of gap-junctional hemichannels. Extracellular calcium-dependent isosmotic volume regulation. 2000 J. Cell Biol. pmid:10704454
Egertová M et al. Fatty acid amide hydrolase expression in rat choroid plexus: possible role in regulation of the sleep-inducing action of oleamide. 2000 Neurosci. Lett. pmid:10713385
Jenkins TC Feeding oleamide to lactating Jersey cows 1. Effects on lactation performance and milk fatty acid composition. 2000 J. Dairy Sci. pmid:10714869
DeLuca DD and Jenkins TC Feeding oleamide to lactating Jersey cows. 2. Effects on nutrient digestibility, plasma fatty acids, and hormones. 2000 J. Dairy Sci. pmid:10750115
Boger DL et al. Exceptionally potent inhibitors of fatty acid amide hydrolase: the enzyme responsible for degradation of endogenous oleamide and anandamide. 2000 Proc. Natl. Acad. Sci. U.S.A. pmid:10805767
Schweitzer JS et al. pH Sensitivity of non-synaptic field bursts in the dentate gyrus. 2000 J. Neurophysiol. pmid:10938317
Bannerman P et al. Early migratory rat neural crest cells express functional gap junctions: evidence that neural crest cell survival requires gap junction function. 2000 J. Neurosci. Res. pmid:10972957